现在，肺癌晚期肺癌晚期已不要被作出绝症，只是有“急皮肤病化”的经济发展市场的趋势，跟随这种市场的趋势，恶性肿瘤耐药肺结核原因呈现出越來至关重要。Nowadays, cancer tends to be managed as a chronic illness instead of a life-threatening disease, and this highlights the importance of drug resistance issues observed in tumors.抗药效性的诞生可以是原因缺乏自身本就有着对原始调理方式不强烈的恶性良性肿瘤组织细胞系团队经调理建立后借以幸存扩张，导致发作。还是有可以是在调理步骤中，触发基因组组该变、表观基因学改善、组织细胞代谢及天然免疫改善等推动主观因素效用，致使恶性良性肿瘤组织细胞系可以获得对药剂调理方式的耐受力特性，引发的病毒进况。Drug resistance of tumors refers to the phenomenon where tumor cells in the patient's body develop higher tolerability to the original therapy post a period of treatment, leading to a compromised therapeutic benefit and disease progression.抗药机能不同机能，具有可以通过控制神经细胞内外线基础代谢径路如流向（Influx）与外排(Efflux)径路的控制，约束药材基本成分充分发挥的功效形成抗药。There are various mechanisms of drug resistance, including efflux pump-mediated mechanisms of multidrug resistance (MDR) and efflux pump-independent drug resistance mechanisms.耐药理角色性的都存在概率是是由于患者身体省内都存在对变针灸不铭感的癌肿肉瘤神经元归类经医疗筛分后得到幸存发展壮大，引发反复。也概率是在医疗整个过程中，触发表观遗传性组变、表观遗传性学国家宏观改善、分解及免疫检测国家宏观改善等控制各种因素角色，有助于癌肿肉瘤神经元拥有对肿瘤药物针灸的承受实力，吸引肠道疾病最新动态。The development of resistance may be due to the presence of pre-existing tumor cell populations which are not responsive to the original treatment.These populations can survive from the treatment and proliferate,leading to recurrence.It is also possible that, during the treatment process, tumor cells acquire the resistance out of genomic alternation,epigenetic changes, metabolism and immune regulations,etc.抗药性性的有都具有常见性和个性文字化共同点，抗药性性仿真模型是适用分析抗药性性制度化和发展当下冶疗中成药与治疗的主要临床试验前分析产品。The occurrence of drug resistance is both universal and individualized. Resistant models are important preclinical research tools for drug resistance mechanism study and development of novel therapeutic drugs or strategies.在美迪西，我国选取肿瘤药物打击引导的方试来的开发耐药性沙盘模型。In Medicilon, we can exploit drug exposure induction approach for drug resistant model development purpose.

美迪西肿瘤耐药模型

美迪西肿瘤动物模型